Keywordsimmunodominant epitope; coronavirus; membrane protein; endodomain; Aachen minipigs (AaMP); non-invasive sampling; porcine cytomegalovirus (PCMV); infection; detection; sensitivity; nested PCR; real-time PCR; porcine epidemic diarrhea virus (PEDV) NJ strain; porcine intestinal epithelial cells; isolation and identification; African swine fever virus; outbreaks; Democratic Republic of Congo; swine; genotypes; molecular epidemiology; p72 gene; p54 gene; CVR; Porcine rotavirus; group A, B, C, E and H rotaviruses; rotavirus vaccines; epidemiology; genetic variability; prevalence; active and passive immunity; swine; zoonotic potential; autophagy machinery; PEDV replication; inflammatory responses; apoptosis; porcine circoviruses; transspecies transmission of viruses; xenotransplantation; virus safety of xenotransplantation; porcine viruses; Pestivirus; classical swine fever; clinical signs; pathogenesis; epidemiology; diagnosis; control; vaccination; marker strategy; Swine influenza; local lung immunity; lung tissue resident memory T cells; universal influenza vaccines; heterosubtypic immunity; PCV2; epidemiology; pigs; vaccination; African swine fever virus; ASFV; virus entry; endocytosis; endosomal pathway; host cell targets; cellular responses; ER stress; apoptosis; autophagy; A179L; PRRSV; genetic diversity; antigenic variation; replication phenotype; neutralization; porcine epidemic diarrhea virus; type II transmembrane serine protease; TMPRSS2; MSPL; virus replication; cell culture-adapted virus; conventional pig; neutralizing antibody; porcine epidemic diarrhea virus; viral shedding; Japanese encephalitis virus; antibody-dependent enhancement of infection; Fc receptor; lentiviral vector vaccine; vaccine-induced protection; persistence; mucosal virus shedding; porcine viruses; PRRSV; HEV; evolution and molecular epidemiology; co-infections; african swine fever virus (ASFV); ASF viral factory; endosomes; endosomal pathway; phosphoinositides; phosphatidylinositol kinases; microtubules; PRRSV; T cell; B cell; NK cell; neutralizing antibody; porcine; memory; adaptive immune response; swine; picornavirus; Seneca Valley virus; emergent disease; vesicular disease; neonatal mortality; classical swine fever virus; virus–cell interactions; attachment; entry; cell apoptosis; virus life cycle
