Reprint

Molecular Mechanism in Epithelial-Mesenchymal Transition (EMT) and Fibrosis

Edited by
March 2024
290 pages
  • ISBN978-3-7258-0702-4 (Hardback)
  • ISBN978-3-7258-0701-7 (PDF)

This book is a reprint of the Special Issue Molecular Mechanism in Epithelial-Mesenchymal Transition (EMT) and Fibrosis that was published in

Biology & Life Sciences
Chemistry & Materials Science
Medicine & Pharmacology
Summary

This Special Issue of the International Journal of Molecular Sciences (IJMS), entitled “Molecular Mechanism in Epithelial-Mesenchymal Transition (EMT) and Fibrosis”, collected 15 original research papers (5 reviews and 10 articles) written by a panel of experts from different countries who highlight recent advances in the EMT process.Navigating the complex field of EMT, this Special Issue introduces the current understanding of the underlying mechanisms of EMT in the evolution and progression of fibrogenesis and discusses potential strategies for attenuating EMT to prevent and/or inhibit fibrosis.Overall, the 15 scientific articles in this Special Issue of the International Journal of Molecular Sciences provide valuable insights into the complex mechanisms governing the EMT process linked to fibrosis and have highlighted the potential of novel therapeutic strategies. In the last few years, the field of EMT has shown considerable promise, and there is still much to be learned. As our understanding continues to grow, we hope that this Special Issue serves as a catalyst for further research and innovation in this developing field.

Format
  • Hardback
License
© 2022 by the authors; CC BY-NC-ND license
Keywords
defense peptides; immune peptides; pulmonary fibrosis; hypersensitivity pneumonitis; extrinsic allergic alveolitis; autoimmune thyroid diseases; Graves’ disease; Hashimoto’s Thyroiditis; TGF-β; EMT; primary cilia; mesenchymal stem cells; mechanotransduction; collagen; oxidation; YAP/TAZ; focal adhesion; proliferative vitreoretinal diseases (PVDs); experimental models; epithelial-mesenchymal transition (EMT); preeclampsia; placenta; EMT; decidua; MET; 15-PGDH; PGT; signal transducer and activator of transcription 3 (STAT3); glaucoma filtration surgery; fibrosis; suppressor of cytokine signaling 3 (SOCS3); epithelial-to-mesenchymal transition; fibrosis; chronic airway diseases; remodelling; fibrosis; collagen 1a1; bleomycin; lansoprazole; Mcp1; Mcp3; Opn; idiopathic pulmonary fibrosis; surfactant related gene mutation; telomere related gene mutation; SFTPC; SFTPA2; RTEL1; TERT; lung tissue; RNA expression; corylin; anti-inflammation; liver fibrosis; hepatic stellate cell; growth arrest-specific gene 6/AXL signaling pathway; inflammation; pulmonary malignancy; epithelial-to-mesenchymal transition; natural products; aggressiveness; tumor stem cells; mechanism of action; idiopathic pulmonary fibrosis; fibroblast; bioinformatics; heterogeneity; predictive model; asthma; airway; fibrosis; epithelial to mesenchymal transition; Cl channel; cell volume; epigenetic; inflammation; fibrosis; DNA methylation; histone modification; ncRNA; STAT4; STAT1; cytokine regulation; interferons; EGF; n/a