KeywordsAnopheles A virus; bunyavirus; Capim virus; Guamá virus; Koongol virus; orthobunyavirus; Tataguine virus; Tete virus; Turlock virus; Witwatersrand virus; Crimean-Congo hemorrhagic fever; viral hemorrhagic fever; reverse genetics; pathogenesis; tick-borne virus; Bunyaviridae; hantavirus; emerging diseases; endonuclease; bunyavirus; high-throughput screening; host-pathogen interaction; haploid genetic screening; RNAi screening; yeast two-hybrid; affinity purification mass spectrometry; bunyavirus; cell entry; endocytosis; hantavirus; RNA virus; tospovirus; virus membrane fusion; virus receptor; Rift Valley fever virus; N-glycosylation; Gn; Gc; sequon; DC-SIGN; L-SIGN; Challenge model; Rift Valley fever; Rift Valley fever virus; Cattle; pathogenicity; Rift Valley fever virus; M-segment; Gn; 78 kD; NSm; precursor; expression strategy; reporter assay; reverse genetics; Bunyaviridae; bunyavirus; nairovirus; Dera Ghazi Khan virus; Erve virus; Ganjam virus; Hughes virus; Qalyub virus; Sakhalin virus; Tunis virus; virus classification; virus taxonomy; phlebovirus; NSs protein; interferon; RIG-I; PKR; Bunyaviridae; phlebovirus; glycoproteins; virus attachment; entry; membrane fusion; signal peptidase; assembly; n/a; Bunyaviridae; hantavirus; virus-host interaction; rodents; humans; insectivores
