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Int. J. Mol. Sci. 2016, 17(3), 429; doi:10.3390/ijms17030429

Caveolae and Caveolin-1 Integrate Reverse Cholesterol Transport and Inflammation in Atherosclerosis

1
School of Pharmacy, Hunan University of Chinese Medicine, Changsha 410208, China
2
Department of Urology, The First Hospital of Hunan University of Chinese Medicine, Changsha 410208, China
3
Department of Pharmacology and Toxicology, University of Mississippi Medical Center, School of Medicine, Jackson, MS 39216, USA
4
Department of Biochemistry & Molecular Biology, the Libin Cardiovascular Institute of Alberta, University of Calgary, 3330 Hospital Drive NW, Calgary, AB T2N 4N1, Canada
*
Author to whom correspondence should be addressed.
Academic Editor: Joseph Moxon
Received: 26 January 2016 / Revised: 16 March 2016 / Accepted: 16 March 2016 / Published: 22 March 2016
(This article belongs to the Section Biochemistry, Molecular and Cellular Biology)
View Full-Text   |   Download PDF [538 KB, uploaded 22 March 2016]   |  

Abstract

Lipid disorder and inflammation play critical roles in the development of atherosclerosis. Reverse cholesterol transport is a key event in lipid metabolism. Caveolae and caveolin-1 are in the center stage of cholesterol transportation and inflammation in macrophages. Here, we propose that reverse cholesterol transport and inflammation in atherosclerosis can be integrated by caveolae and caveolin-1. View Full-Text
Keywords: caveolae; caveolin-1; reverse cholesterol transport; inflammation; atherosclerosis caveolae; caveolin-1; reverse cholesterol transport; inflammation; atherosclerosis
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This is an open access article distributed under the Creative Commons Attribution License which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. (CC BY 4.0).

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MDPI and ACS Style

Qin, L.; Zhu, N.; Ao, B.-X.; Liu, C.; Shi, Y.-N.; Du, K.; Chen, J.-X.; Zheng, X.-L.; Liao, D.-F. Caveolae and Caveolin-1 Integrate Reverse Cholesterol Transport and Inflammation in Atherosclerosis. Int. J. Mol. Sci. 2016, 17, 429.

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