Fe Deficiency, Dietary Bioavailability and Absorption

Edited by
September 2018
214 pages
  • ISBN978-3-03897-230-3 (Paperback)
  • ISBN978-3-03897-231-0 (PDF)

This book is a reprint of the Special Issue Fe Deficiency, Dietary Bioavailbility and Absorption that was published in

Medicine & Pharmacology
Public Health & Healthcare
The World Health Organization (WHO) estimates that approximately one-third of worldwide infant deaths, and one half in developing countries, can be attributed to malnutrition. More specifically, iron (Fe) deficiency is the most common nutritional deficiency worldwide and a major cause of infant mortality. Fe deficiency is particularly widespread in low-income countries because of a general lack of consumption of animal products (which can promote non-heme Fe absorption and contain highly bioavailable heme Fe) coupled with a high consumption of a monotonous diet of cereal grains and legumes. Such diets are low in bioavailable Fe due to the presence of phytic acid and certain polyphenols that are inhibitors of Fe bioavailability. Diets with chronically poor Fe bioavailability which result in high prevalence of Fe deficiency and anemia, increase the risk of all-cause child mortalities and also may lead to many pathophysiological consequences including stunted growth, low birth weight, delayed mental development and motor functioning, among others. Thus, a crucial step in alleviating Fe deficiency anemia is through understanding how specific dietary practices and components contribute to the Fe status in a particular region where Fe deficiency is prevalent. The aim of this Special Issue is to report on the recent advances and research developments related to the improvements of dietary Fe bioavailability and absorption in an effort to alleviate dietary Fe deficiency.
  • Paperback
© 2019 by the authors; CC BY license
anemia; cluster-randomized controlled trial; complementary food; Côte d’Ivoire; infant cereal; iron deficiency; iron fortification; Plasmodium falciparum; sodium iron EDTA; beans; Phaseolus vulgaris; iron; bioavailability; biofortification; iron related-hormone; endurance training; iron supplementation; lentil; iron; fortification; NaFeEDTA; FeSO4·7H2O; FeSO4·H2O; iron bioavailability; iron fortification; simulated gastrointestinal digestion; anaemia; Crohn’s disease; IBD; iron deficiency; therapy; ulcerative colitis; iron metabolism; anemia; endurance; exercise; sport performance; iron; iron deficiency; iron supplementation; infants; children; neurodevelopment; brain dysfunction; lentil; iron; fortification; bioavailability; Bangladesh; ferritin; hemodialysis; hepcidin-25; inflammation; iron deficiency anemia; oral iron therapy; sickle cell anemia; hepcidin; iron overload; chronic kidney disease; ferritin; C-reactive protein; hepcidin; inflammation; iron deficiency anemia