Keywordsfungal pathogenesis; Candida albicans; oxidative stress; stress signaling; advanced glycation end products; oxidative stress; type 2 diabetes mellitus; insulin resistance; β cell dysfunction; diabetic complications; oxidative stress; NADPH oxidase; peroxiredoxin; protein disulfide isomerase; superoxide radical anion; hydrogen peroxide; hydroxyl radical; mitochondria; integral membrane reductases; lignin degradation; skeletal muscle; ROS; exercise; mitochondria; force generation; antioxidants; PGC-1α; oxidative post-translational modification; inflammation; cardiovascular disease; protein carbonyls; nitrotyrosine; chlorotyrosine; LC-MS/MS; precursor ion scanning; neutral loss scanning; multiple reaction monitoring; superoxide radical; hydrogen peroxide; hydroxyl radical; nitric oxide; mitochondria; lipofuscin; AGEs; cornified envelope; calcium gradient; extrinsic and intrinsic aging; DNA damage; lipid peroxidation; antioxidants; carbonylation; heme oxygenase activity; bilirubin; hemin; endoplasmic reticulum; mitochondria; neutrophils; NETs; autophagy; NADPH oxidase; MAPK/ERK; inflammation; iron; oxidative stress; metabolism; haskap; inflammation; cytokines; polyphenols; macrophages; functional food; white adipose tissue; adipocytes; hypoxia; oxidative stress; cell metabolism; oxygen radicals; ROS; proteomics; mass spectrometry; electron spin resonance spectroscopy; respirometry; redox homeostasis; aging; photosynthesis; adaptation; high-resolution respirometry; H2O2 flux; Amplex Red; HEK 293T; mouse brain homogenate; mouse cardiac mitochondria; Maxi calcium-activated potassium ion channels; BK channels; KCNMA1; redox mechanisms; oxidation; hypoxia; ischemia; oxidative stress; reactive oxygen species; metabolic damage; metabolite repair; enzyme promiscuity; underground metabolism; 4-hydroxy-nonenal; HNE; n-6 polyunsaturated fatty acids; Michael adducts; mercapturic acid
