KeywordsDNA damage; DNA protection; DNA repair; vitamin C; vitamin C transport; sodium-dependent vitamin C transporters, SVCT1 and SVCT2; glucose transporters (GLUTs); regulation of transport; vitamin C; sepsis; neutrophils; NETosis; cell free DNA; nuclear factor κB; l-ascorbic acid; ascorbate; photosynthesis; DHAR; MDAR; reactive oxygen species; high concentrations of vitamin C; pharmacogenomics; pharmacoproteomics; haptoglobin; iron; polymorphism; scurvy; vitamin C; ascorbate; ascorbic acid; human; plasma; urine; semen; leukocytes; skeletal muscle; ascorbate; dietary vitamin C; bioavailability; animal studies; human studies; bioflavonoids; human; ascorbic acid; ascorbate; plasma; urine; kiwi fruit; vitamin C; ascorbic acid; cell culture; animals; human; study design; ascorbic acid; diabetes mellitus; oxidative stress; apoptosis; proinflammatory; cytokine; sodium-dependent vitamin C transporter 2 (SVCT2); glucose transporter 1 (GLUT1); rat; streptozotocin; middle cerebral artery occlusion and reperfusion; ascorbic acid; vitamin C; brain; cognitive function; alzheimer’s disease; dementia; aging; elderly; endothelial function; blood-brain barrier; SVCT (sodium-dependent vitamin C transporter)
