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Correction

Correction: Schildgen, V., et al. Human Bocavirus Infection of Permanent Cells Differentiated to Air-Liquid Interface Cultures Activates Transcription of Pathways Involved in Tumorigenesis. Cancers 2018, 10, 410

1
Kliniken der Stadt Köln gGmbH, Institut für Pathologie, Kliniken der Privaten Universität Witten/Herdecke mit Sitz in Köln, Ostmerheimer Str. 200, D-51109 Köln/Cologne, Germany
2
Universität Witten/Herdecke, Lehrstuhl für Biologische und Materialkundliche Grundlagen der Zahnmedizin, D-58448 Witten, Germany
*
Authors to whom correspondence should be addressed.
Cancers 2021, 13(8), 1774; https://doi.org/10.3390/cancers13081774
Submission received: 2 February 2021 / Accepted: 25 March 2021 / Published: 8 April 2021
The authors wish to make the following correction to this paper [1]: In the published version, Figure 3b appeared as a duplication of Figure 3a, while the figure legend correctly described Figure 3b with different content.
The original version of Figure 3 that has to be corrected is:
Figure 3. Immunohistochemical staining of CuFi-8 air–liquid-interface cultures. (a) Scheme 8. cells and HBoV-positive lung tumor biopsies, whereas mock-infected CuFi-8 cultures, as well as HBoV-negative lung tumors, were CEA-negative. CuFi-1 and Cufi-5 cells were not CEA-positive at all. (b) PAS–Alcian blue staining reveals higher production of acid mucins in CuFi-8 cells compared to those in CuFi-1 and CuFi-5 cells in general. Beyond that, there is an increased expression of acid mucins after HBoV infection in CuFi-8 cells.
Figure 3. Immunohistochemical staining of CuFi-8 air–liquid-interface cultures. (a) Scheme 8. cells and HBoV-positive lung tumor biopsies, whereas mock-infected CuFi-8 cultures, as well as HBoV-negative lung tumors, were CEA-negative. CuFi-1 and Cufi-5 cells were not CEA-positive at all. (b) PAS–Alcian blue staining reveals higher production of acid mucins in CuFi-8 cells compared to those in CuFi-1 and CuFi-5 cells in general. Beyond that, there is an increased expression of acid mucins after HBoV infection in CuFi-8 cells.
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It should be replaced with the following Figure 3:
We stress that these errors were purely due to human error and oversight; all corrections done do not affect or change the written proportion of the figure legend, interpretation of the results, or the final conclusions of this manuscript. The manuscript will be updated. The authors would like to apologize for any inconvenience caused. All changes have been reviewed and verified by the Academic Editors.

Author Contributions

M.P. performed cell culturing, extraction of nucleic acids, generation of infectious virus and infection of air–liquid interface cultures. S.K. performed immunohistochemistry and standard histological stainings and analyzed the respective results. W.H.A. performed electron microscopy and analyzed the respective results. V.S. and O.S. analyzed the transcriptome data, performed the IPA analyses, interpreted the data, supervised the entire study, and wrote the manuscript. All authors have read and agreed to the published version of the manuscript.

Funding

This work was supported by an intramural research grant from the Private University of Witten/Herdecke to V.S., a research grant from the Beatrix-Lichtken-Stiftung (Cologne, Germany) to O.S., and a research grant from the Lörcher-Stiftung (Cologne/Frechen, Germany) to O.S.

Conflicts of Interest

The authors declare no conflict of interest.

Reference

  1. Schildgen, V.; Pieper, M.; Khalfaoui, S.; Arnold, W.H.; Schildgen, O. Human Bocavirus Infection of Permanent Cells Differentiated to Air-Liquid Interface Cultures Activates Transcription of Pathways Involved in Tumorigenesis. Cancers 2018, 10, 410. [Google Scholar] [CrossRef] [PubMed] [Green Version]
Figure 3. Immunohistochemical staining of CuFi-8 air–liquid-interface cultures. (a) Scheme 8. cells and HBoV-positive lung tumor biopsies, whereas mock-infected CuFi-8 cultures, as well as HBoV-negative lung tumors, were CEA-negative. CuFi-1 and Cufi-5 cells were not CEA-positive at all. (b) PAS–Alcian blue staining reveals higher production of acid mucins in CuFi-8 cells compared to those in CuFi-1 and CuFi-5 cells in general. Beyond that, there is an increased expression of acid mucins after HBoV infection in CuFi-8 cells.
Figure 3. Immunohistochemical staining of CuFi-8 air–liquid-interface cultures. (a) Scheme 8. cells and HBoV-positive lung tumor biopsies, whereas mock-infected CuFi-8 cultures, as well as HBoV-negative lung tumors, were CEA-negative. CuFi-1 and Cufi-5 cells were not CEA-positive at all. (b) PAS–Alcian blue staining reveals higher production of acid mucins in CuFi-8 cells compared to those in CuFi-1 and CuFi-5 cells in general. Beyond that, there is an increased expression of acid mucins after HBoV infection in CuFi-8 cells.
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MDPI and ACS Style

Schildgen, V.; Pieper, M.; Khalfaoui, S.; Arnold, W.H.; Schildgen, O. Correction: Schildgen, V., et al. Human Bocavirus Infection of Permanent Cells Differentiated to Air-Liquid Interface Cultures Activates Transcription of Pathways Involved in Tumorigenesis. Cancers 2018, 10, 410. Cancers 2021, 13, 1774. https://doi.org/10.3390/cancers13081774

AMA Style

Schildgen V, Pieper M, Khalfaoui S, Arnold WH, Schildgen O. Correction: Schildgen, V., et al. Human Bocavirus Infection of Permanent Cells Differentiated to Air-Liquid Interface Cultures Activates Transcription of Pathways Involved in Tumorigenesis. Cancers 2018, 10, 410. Cancers. 2021; 13(8):1774. https://doi.org/10.3390/cancers13081774

Chicago/Turabian Style

Schildgen, Verena, Monika Pieper, Soumaya Khalfaoui, Wolfgang H. Arnold, and Oliver Schildgen. 2021. "Correction: Schildgen, V., et al. Human Bocavirus Infection of Permanent Cells Differentiated to Air-Liquid Interface Cultures Activates Transcription of Pathways Involved in Tumorigenesis. Cancers 2018, 10, 410" Cancers 13, no. 8: 1774. https://doi.org/10.3390/cancers13081774

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