Daniel F Wallace received his BSc (Hons) in Biochemistry from Sheffield University in 1994 and his Ph.D. in Genetics from University College London in 1999. He is now an Associate Professor at the Queensland University of Technology (QUT). Dr Wallace’s main research interests lie in understanding the molecular bases underlying the control of iron homeostasis and iron-associated disorders. He has published extensively in the fields of HFE and non-HFE haemochromatosis genetics, cell biology and the regulation of iron metabolism. His research over the past 20 years has been instrumental in defining the genetic causes of iron overload and the role of the liver and hepcidin in regulating body iron balance. He is also interested in how cancers exploit the iron regulatory machinery to increase iron availability for their growth and following from the development of future therapeutics for cancer.
V. Nathan Subramaniam is an NHMRC Senior Research Fellow and Research Capacity Building Professor in the School of Biomedical Sciences, QUT. Prof Nathan Subramaniam received his PhD from Purdue University, W. Lafayette, Indiana, USA. He then joined the group of Prof Wanjin Hong at the Institute of Molecular and Cell Biology, Singapore, where he completed his postdoctoral training on cell biology and protein trafficking. Prof Nathan Subramaniam established his research group, the Membrane Transport Laboratory at the Queensland Institute of Medical Research, Brisbane, Australia in 1999. In 2016, he was appointed a Research Capacity Building Professor as Professor in Biomedical Sciences (Molecular Medicine) at the Queensland University of Technology. He established his research group, the Hepatogenomics Research Group, at the School of Biomedical Sciences, Institute of Health and Biomedical Innovation in August 2016. Prof Subramaniam's interests lie in the study of liver injury and understanding how the liver regulates iron homeostasis. His basic science interests include defining the functional consequences of these disease-causing mutations and elucidating the molecules and mechanisms regulating iron homeostasis.
