Dr. Malik Bisserier is an assistant professor of Cell Biology and
Anatomy and of Physiology at New York Medical College, with over 10 years of
experience in biotechnology, molecular biology, and epigenetics. He received
his Ph.D. from the University of Toulouse in France, followed by two years of
postdoctoral training in pathology at Yale University. He joined NYMC from the
Cardiovascular Research Institute at the Icahn School of Medicine at Mount
Sinai in New York City. His postdoctoral work uncovered a new epigenetic
mechanism underlying the loss of BMPR2 in pulmonary arterial hypertension. His
work has been published in high-impact journals, including Circulation,
Circulation Research, Cardiovascular Research, Blood, Journal of the American
College of Cardiology, and Molecular Therapy. He is a member of the American
Thoracic Society, the American Heart Association, the International Society of Heart
Research, and the Pulmonary Vascular Research Institute, and he currently serves
as a guest editor of several journals. He has been awarded several prestigious
recognitions, including the Cournand and Comroe Early Career Investigator Award
from the American Heart Association.
Lahouaria Hadri graduated from the University of Paris XI, Faculty of Pharmacy, Châtenay-Malabry, in 2005. In 2006, she relocated to the United States and undertook a postdoc position at Massachusetts General Hospital, Harvard Medical School, in Boston, and in 2007, she moved to Icahn School of Medicine at Mount Sinai in New York City with the team. The Hadri Lab focuses on cardiovascular diseases, pulmonary arterial hypertension (PAH), and idiopathic pulmonary fibrosis (IPF). Its current research objectives are to provide an in-depth analysis of calcium-handling proteins’ roles and their cellular signaling networks in cardiovascular and lung diseases and to define a platform for the design of innovative therapeutic strategies using gene- and drug-targeted therapies. The lab also has a particular interest in exploring the role of epigenetic modifications and SIN3a/histone deacetylase complex in various cardiopulmonary diseases.
