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Open AccessArticle

Brugada-Like Electrocardiographic Patterns Induced by Hyperkalemia

1
Department of Intensive Care, Republican Hospital of Kaunas
2
Department of Intensive Care, Medical Academy, Lithuanian University of Health Sciences
3
Department of Anesthesiology, Medical Academy, Lithuanian University of Health Sciences, Lithuania
*
Author to whom correspondence should be addressed.
Medicina 2013, 49(3), 24; https://doi.org/10.3390/medicina49030024
Received: 21 May 2012 / Accepted: 30 March 2013 / Published: 4 April 2013
Brugada syndrome was described in 1992 as a new clinical and electrocardiographic syndrome involving susceptibility to ventricular arrhythmias and sudden cardiac death in patients with no obvious structural heart disease. Brugada syndrome is characterized by a hereditary anomaly in the sodium ion channel (mutation of the SCN5A gene) identified by a wide QRS associated with the ST-segment elevation and the T‑wave inversion in the right precordial leads. The Brugada-like electrocardiographic pattern can be caused by sodium channel-blocking drugs and electrolyte disorders. Hyperkalemia may produce multiple ECG abnormalities, including the ST-segment elevation and pseudomyocardial infarction with a resolution of these abnormalities after the correction of hyperkalemia. This article describes 8 cases of pseudoanteroseptal myocardial infarction in acute renal insufficiency with hyperkalemia. The ST-segment elevation related to hyperkalemia is resolved by the reduced serum potassium level. Clinicians should recognize that hyperkalemia is one of the etiologies of the Brugada-like electrocardiographic pattern.
Keywords: Brugada syndrome; Brugada electrocardiographic pattern; electrocardiographic hyperkalemia signs Brugada syndrome; Brugada electrocardiographic pattern; electrocardiographic hyperkalemia signs
MDPI and ACS Style

Reingardienė, D.; Vilčinskaitė, J.; Bilskienė, D. Brugada-Like Electrocardiographic Patterns Induced by Hyperkalemia. Medicina 2013, 49, 24.

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