Special Issue "Wound Healing and Tissue Regeneration"

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A special issue of Journal of Developmental Biology (ISSN 2221-3759).

Deadline for manuscript submissions: closed (31 October 2015)

Special Issue Editor

Guest Editor
Dr. Robin C. Muise-Helmericks
Department of Regenerative Medicine and Cell Biology, Medical University of South Carolina, Charleston SC, 29425, USA
Website: http://cba.musc.edu/Faculty/Muise-HelmericksR.htm
Phone: 843-792-4760
Interests: Akt family of kinases; angiogenesis; wound healing; mitochondrial function and control

Special Issue Information

Submission

Manuscripts should be submitted online at www.mdpi.com by registering and logging in to this website. Once you are registered, click here to go to the submission form. Manuscripts can be submitted until the deadline. Papers will be published continuously (as soon as accepted) and will be listed together on the special issue website. Research articles, review articles as well as communications are invited. For planned papers, a title and short abstract (about 100 words) can be sent to the Editorial Office for announcement on this website.

Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). All manuscripts are refereed through a peer-review process. A guide for authors and other relevant information for submission of manuscripts is available on the Instructions for Authors page. Journal of Developmental Biology is an international peer-reviewed Open Access quarterly journal published by MDPI.

Please visit the Instructions for Authors page before submitting a manuscript. For the first couple of issues the Article Processing Charge (APC) will be waived for well-prepared manuscripts. English correction and/or formatting fees of 250 CHF (Swiss Francs) will be charged in certain cases for those articles accepted for publication that require extensive additional formatting and/or English corrections.

Published Papers (6 papers)

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Displaying article 1-6
n° 1
by  and
J. Dev. Biol. 2016, 4(1), 1; doi:10.3390/jdb4010001
Received: 2 November 2015 / Revised: 4 December 2015 / Accepted: 17 December 2015 / Published: 23 December 2015
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(This article belongs to the Special Issue Wound Healing and Tissue Regeneration)
p. 71-89
by
J. Dev. Biol. 2015, 3(2), 71-89; doi:10.3390/jdb3020071
Received: 13 March 2015 / Revised: 4 May 2015 / Accepted: 6 May 2015 / Published: 12 May 2015
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(This article belongs to the Special Issue Wound Healing and Tissue Regeneration)
p. 57-70
by  and
J. Dev. Biol. 2015, 3(2), 57-70; doi:10.3390/jdb3020057
Received: 30 December 2014 / Revised: 4 April 2015 / Accepted: 21 April 2015 / Published: 27 April 2015
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(This article belongs to the Special Issue Wound Healing and Tissue Regeneration)
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p. 11-24
by  and
J. Dev. Biol. 2015, 3(1), 11-24; doi:10.3390/jdb3010011
Received: 13 January 2015 / Revised: 10 February 2015 / Accepted: 12 February 2015 / Published: 2 March 2015
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(This article belongs to the Special Issue Wound Healing and Tissue Regeneration)
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p. 198-209
by , ,  and
J. Dev. Biol. 2014, 2(4), 198-209; doi:10.3390/jdb2040198
Received: 13 October 2014 / Revised: 7 November 2014 / Accepted: 10 December 2014 / Published: 19 December 2014
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(This article belongs to the Special Issue Wound Healing and Tissue Regeneration)
p. 210-229
by
J. Dev. Biol. 2014, 2(4), 210-229; doi:10.3390/jdb2040210
Received: 28 October 2014 / Revised: 24 November 2014 / Accepted: 9 December 2014 / Published: 19 December 2014
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(This article belongs to the Special Issue Wound Healing and Tissue Regeneration)
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Planned Papers

The below list represents only planned manuscripts. Some of these manuscripts have not been received by the Editorial Office yet. Papers submitted to MDPI journals are subject to peer-review.

Type of Paper: Review
Title: Beyond the mammalian heart: fish and amphibians as a model for cardiac repair and regeneration
Authors: Dr. Kelly McLaughlin et al.
Affiliation: Department of Biology, Tufts University, Medford, Massachusetts, USA
Abstract: While mammals have long been the model organisms of choice for studying heart damage, recent studies in fish and amphibians have shown these animals to possess far more robust abilities in cardiac repair and regeneration. These abilities make the so-called lower vertebrates prime candidates for discovering novel strategies to repair cardiac damage. Our review will focus on the current state of research into how these animals perform this remarkable feat of regeneration, as well as the array of tools and techniques available to study them.

Type of Paper: Review
Title: The role of TGF-beta signalling in wound healing and tissue regeneration
Authors: Richard W.D. Gilbert 1, Matthew Vickaryous 2, Alicia M. Viloria-Petit 2
Affiliation: 1 School of Medicine, University College Cork, Cork, Ireland; 2 Department of Biomedical Sciences, Ontario Veterinary College, University of Guelph, Guelph, Ontario, Canada
Abstract: Following complex tissue injury a series of coordinated biological events leads to the eventual repair of the damaged structures, resulting in either scar formation or tissue regeneration. In most examples of mammalian wound healing, response to injury involves scar formation and often, functional impairment. Interestingly, numerous other vertebrate species possess varied abilities to regenerate injured tissues and structures resulting in scar free tissue repair. Understanding the molecular pathways that enable and favor tissue regeneration over scar formation has the potential to yield great medical benefit. Amongst the factors that determine this balance, several key developmental signalling pathways have been implicated. Here we review the role of the transforming growth factor beta (TGF-β) superfamily in tissue repair. We discuss the role of these growth factors and their key downstream signalling mediators in determining the balance between scar formation and tissue regeneration.

Last update: 13 October 2015

J. Dev. Biol. EISSN 2221-3759 Published by MDPI AG, Basel, Switzerland RSS E-Mail Table of Contents Alert