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Viruses 2017, 9(7), 171; doi:10.3390/v9070171

Complex Virus–Host Interactions Involved in the Regulation of Classical Swine Fever Virus Replication: A Minireview

State Key Laboratory of Veterinary Biotechnology, Harbin Veterinary Research Institute, Chinese Academy of Agricultural Sciences, 678 Haping Road, Harbin 150069, China
S.L. and J.W. contributed equally to this article.
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Author to whom correspondence should be addressed.
Academic Editors: Linda Dixon and Simon Graham
Received: 24 April 2017 / Revised: 15 June 2017 / Accepted: 27 June 2017 / Published: 5 July 2017
(This article belongs to the Special Issue Porcine Viruses)
View Full-Text   |   Download PDF [4128 KB, uploaded 5 July 2017]   |  

Abstract

Classical swine fever (CSF), caused by classical swine fever virus (CSFV), is one of the most devastating epizootic diseases of pigs in many countries. Viruses are small intracellular parasites and thus rely on the cellular factors for replication. Fundamental aspects of CSFV–host interactions have been well described, such as factors contributing to viral attachment, modulation of genomic replication and translation, antagonism of innate immunity, and inhibition of cell apoptosis. However, those host factors that participate in the viral entry, assembly, and release largely remain to be elucidated. In this review, we summarize recent progress in the virus–host interactions involved in the life cycle of CSFV and analyze the potential mechanisms of viral entry, assembly, and release. We conclude with future perspectives and highlight areas that require further understanding. View Full-Text
Keywords: classical swine fever virus; virus–cell interactions; attachment; entry; cell apoptosis; virus life cycle classical swine fever virus; virus–cell interactions; attachment; entry; cell apoptosis; virus life cycle
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Li, S.; Wang, J.; Yang, Q.; Naveed Anwar, M.; Yu, S.; Qiu, H.-J. Complex Virus–Host Interactions Involved in the Regulation of Classical Swine Fever Virus Replication: A Minireview. Viruses 2017, 9, 171.

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